The study examines how the SnRK1 catalytic subunit KIN10 integrates carbon availability with root growth regulation in Arabidopsis thaliana. Loss of KIN10 reduces glucose‑induced inhibition of root elongation and triggers widespread transcriptional reprogramming of metabolic and hormonal pathways, notably affecting auxin and jasmonate signaling under sucrose supplementation. These findings highlight KIN10 as a central hub linking energy status to developmental and environmental cues in roots.

The study compared physiological, ion‑balance, and metabolic responses of two maize inbred lines—salt‑sensitive C68 and salt‑tolerant NC326—under salinity stress. Untargeted metabolomics identified 56 metabolites and, together with genetic analysis, linked 10 candidate genes to key protective metabolites, revealing constitutive and inducible mechanisms of salt tolerance.

The study models maize flowering time plasticity using a physiological reaction norm derived from multi-environment trial data, revealing genotype-specific differences in temperature-driven development and photoperiod perception. It introduces an envirotyping metric that shows genotypes can experience markedly different photoperiods even within the same environment, and demonstrates distinct adaptive strategies between tropical and temperate germplasm.

The study generated a temporal physiological and metabolomic map of leaf senescence in diverse maize inbred lines differing in stay‑green phenotype, identifying 84 metabolites associated with senescence and distinct metabolic signatures between stay‑green and non‑stay‑green lines. Integration of metabolite data with genomic information uncovered 56 candidate genes, and reverse‑genetic validation in maize and Arabidopsis demonstrated conserved roles for phenylpropanoids such as naringenin chalcone and eriodictyol in regulating senescence.